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Asymptomatic covid patients with no symptoms still get lung damage

Results from China, Japan, and USA show that half or more of asymptomatic cases have lung damage

Asymptomatic cases show a weaker immune response. In a small study in China, the asymptomatic cases had a lower, slower immune response, which means they shed virus for longer than the symptomatic cases, perhaps even as long as 14 days. Though the presence of viral RNA does not necessarily mean they can infect other people. It may be fragments of the whole virus.

Covid lung CT scan

a, CT scan of a 45-year-old female showing focal ground-glass opacities in the lower lobe of the left lung (arrow). b, CT scan of a 50-year-old female showing ground-glass opacities and stripes coexisting in the lower lobe of the right lung (arrows).

Below, a Scripps review found the rate of lung damage was as high as 70% of asymptomatic cases. Both these studies are small, non randomized, retrospective studies.

It’s not surprising that people might not notice that their lungs were underperfoming. We have so much extra lung capacity for day to day living that many people don’t notice damage.  In cases of lung cancer, people are often not diagnosed until they are in an advanced late stage.  Presumably, humans hardly ever push their lungs to the limit now, so we don’t notice if they are not performing at peak.

What Does Asymptomatic COVID-19 Look Like Under the Surface?

Molly Walker, MedPage

Asymptomatic individuals carrying SARS-CoV-2 shed the virus longer than those with COVID-19 symptoms, with other lab findings suggesting the symptomatic patients mounted more robust immune responses, a small study in China found.

Virus-specific IgG antibody titers and cytokine levels were also significantly lower among asymptomatic patients in the acute phase of infection, when viral RNA can be found in respiratory specimens, the authors wrote in Nature Medicine — both of which indicated that immune responses weren’t as strong in the asymptomatic group.

For the study, they examined data from 178 patients with PCR-confirmed SARS-CoV-2 infection in the Wanzhou District in China, including 37 without symptoms. Median age in the latter was 41, and 22 were women.

Lab tests show there was lung damage in just over half the group, inflammation (CRP), and there was some liver damage (ALT):

Lab values and imaging were not entirely normal for the asymptomatic group. Eleven had increased C-reactive protein levels (CRP) and six had elevated levels of alanine aminotransferase (ALT). Chest CT found “focal ground-glass opacities” in 11 and “stripe shadows and/or diffuse consolidation” in another 10 of the group; in two-thirds of these 21 patients, the abnormalities were in only one lung. The remaining 16 showed entirely normal imaging.

Antibody levels fell faster in asymptomatic people

In the early convalescent phase, defined as 8 weeks after hospital discharge, symptomatic patients had higher IgG levels, though both groups experienced over 90% decreases in IgG levels. A larger proportion of asymptomatic patients had decreases in neutralizing serum antibody levels versus symptomatic patients (81.1% vs 62.2%, respectively).

These findings should serve as a caution against assuming prior infection confers immunity to future infection, Qiu and colleagues said.

A Scripps study out a few days ago also suggests in people with no symptoms, Coronavirus may be damaging lungs. When they did a CT scan of asymptomatic people on the Diamond Princess, as many as 70% showed some lung damage.

CT Scans show lung injury in asymptomatic carriers of Covid-19

The authors also conclude that the absence of symptoms may not imply an absence of harm. CT scans conducted on 54% of 76 asymptomatic individuals on the Diamond Princess cruise ship, appear to show significant subclinical lung abnormalities, raising the possibility of SARS-CoV-2 infection impacting lung function that might not be immediately apparent. The scientists say further research is needed to confirm the potential significance of this finding.

CT = Computed Tomography.

They estimate up to half of Covid patients may be asymptomatic, though this is contested. A big problem with estimating the rate of asymptomatic infections is that very few studies follow the patients for two weeks to find out if they do develop symptoms.  Asymptomatic cases may be just presymptomatic. Another recent review considered only studies with follow up, and pooled the results to find only 15% were true cases of asymptomatic infection. (More on that later).

REFERENCES

Long QX, et al “Clinical and immunological assessment of asymptomatic SARS-CoV-2 infections” Nat Med 2020; DOI: 10.1038/s41591-020-0965-6.

Ran and Topal, (2020) Annals of Internal Medicine.

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Rating: 7.5/10 (39 votes cast)
Asymptomatic covid patients with no symptoms still get lung damage, 7.5 out of 10 based on 39 ratings

66 comments to Asymptomatic covid patients with no symptoms still get lung damage

  • #
    MrGrimNasty

    Hardly surprising is it – it’s the same with most viral infections that you hardly notice, probably happens to everyone many times a year. If they check for myositis they’ll find that too. It’s all perfectly normal stuff! Is calling a normal effect of the immune system function a lung injury or damage supposed to make it scary or something?

    Every one will recall days/periods when they felt slightly off but not really ill for a few days or weeks, we dismiss it as working too hard or not enough sleep or stress. If you do a physical job or regular exercise you notice this especially. That thing you did easily last week suddenly makes you slightly out of breathe or that thigh exercise seems like unusually hard work or you just can’t get within a minute of your normal 5k time. Then one day you wake up and it’s like you’re re-energized, and only then you twig you must have had a bit of a bug.

    It’s still just bad flu!

    https://erj.ersjournals.com/content/45/5/1463

    467

    • #
      Bill In Oz

      I say your comment is BS.
      It’s a killer MGN.
      PS Your link is from 2015.
      Wonder what the authors think now. ?

      325

    • #
      Sceptical Sam

      It’s still just bad flu!

      Except it’s not the “flu”.

      After all the evidence that’s been presented you still hold to your preconceived and irrational ideas.

      Totally unscientific. Why are you here?

      And that applies to your 36 thumbers who agree with you. No wonder the world is in such a bad state.

      43

      • #
        Bill In Oz

        Yes Sam, being sceptical in science is a necessary part of a scientific approach to life.
        But with disease rejecting the evidence because personal (non medical ) prejudices is bizarre and has no place on this blog..

        This is an immediate issue of life and death for many people that this virus infects.
        For many others, as Jo’s post demonstrates with evidence, it isa matter of significant damage for a long time.

        16

      • #

        What about the Spanish flu. Was that not a flu either?

        The most distinguishing criteria separating a respiratory viral cold from a viral flu is a fever. What about the asymptomatic cases where no fever is present. Does this make the China virus just a viral cold?

        Any flu (or cold) can have asymptomatic carriers, be deadly to people with comorbidities and cause lung damage, so what’s the criteria for claiming that the China virus is not a flu?

        42

    • #
      Evidence Please

      It’s the asymptomatic cases WITH symptoms that I worry about

      13

      • #
        Kalm Keith

        You are symptomatic of the modern “look at me I’m a genius” syndrome.

        Without evidence of independent brain function.
        :-)

        20

    • #
      Bulldust

      Imagine my shock that X-rays of Chinese people show lung damage.

      Let’s see a control group to compare. I visited China a few years back … went to the Great Wall and couldn’t see more than about 100 feet in any direction. And we are surprised Chinese people have poor lung function?

      71

    • #
      TedM

      Copied and pasted from the link you provided.

      “Influenza-mediated damage of the airway, alveolar epithelium and alveolar endothelium results from a combination of: 1) intrinsic viral pathogenicity, attributable to its tropism for host airway and alveolar epithelial cells; and 2) a robust host innate immune response, which, while contributing to viral clearance, can worsen the severity of lung injury.”

      This is totally unrelated to asymptomatic infections.

      I suspect that you have recruited some green tickers.

      00

  • #
    DonK31

    “In the early convalescent phase, defined as 8 weeks after hospital discharge, symptomatic patients had higher IgG levels, though both groups experienced over 90% decreases in IgG levels. A larger proportion of asymptomatic patients had decreases in neutralizing serum antibody levels versus symptomatic patients (81.1% vs 62.2%, respectively).”

    I am confused. Are they comparing symptomatic people who just got out of the hospital with asymptomatic people who just got out of the hospital? If one is asymptomatic, why has he been in the hospital? If they are not comparing like subjects, it like comparing apples and bananas.
    If an asymptomatic person hasn’t been in a hospital, how do the researchers know how far into convalescence he is?

    240

    • #
      Broadie

      ‘comparing apples & bananas’
      The new normal in studies. No longer do we compare results to those of a control group.
      If I was looking at Ruby Princess passengers, I would be comparing the post voyage mortality and viral infection rates to similar cruises in similar weather conditions over previous years. The weather that cruise went through was shocking from my recollection and sea sickness and confinement would be a test for anyone let alone the age demographic of those scenic destination cruises.

      122

  • #
    dinn, rob

    ho ho, Shi Zhengli tells us of combining hiv-system and ACE2 in bats
    (6-20-20 Brazil 55.2/417= 13% increase/day new cases/active cases https://www.worldometers.info/coronavirus/country/brazil
    Mexico 566/1695= 33% increase/day https://www.worldometers.info/coronavirus/country/mexico
    Chile 63/295= 21% increase/day https://www.worldometers.info/coronavirus/country/chile
    India 14.7/154= 9.5% increase/day https://www.worldometers.info/coronavirus/country/india/
    South Africa 38/342= 11% increase/day
    ………………………………)
    Feb 2008 article of Shi Zhengli team: combining hiv system with cell lines expressing ACE2 of human, civet or horseshoe bat
    Difference in Receptor Usage between Severe Acute Respiratory Syndrome (SARS) Coronavirus and SARS-Like Coronavirus of Bat Origin
    Wuze Ren, Xiuxia Qu, Wendong Li, Zhenggang Han, Meng Yu, Peng Zhou, Shu-Yi Zhang, Lin-Fa Wang, Hongkui Deng, Zhengli Shi
    In this study we investigated the receptor usage of the (SL=Sars-related) SL-CoV S by combining a human immunodeficiency virus-based pseudovirus system with cell lines expressing the ACE2 molecules of human, civet or horseshoe bat. In addition to full-length S of SL-CoV and SARS-CoV, a series of S chimeras was constructed by inserting different sequences of the SARS-CoV S into the SL-CoV S backbone. Several important observations were made from this study. First, the SL-CoV S was unable to use any of the three ACE2 molecules as its receptor. Second, the SARS-CoV S failed to enter cells expressing the bat ACE2. Third, the chimeric S covering the previously defined receptor-binding domain gained its ability to enter cells via human ACE2, albeit with different efficiencies for different constructs. Fourth, a minimal insert region (amino acids 310 to 518) was found to be sufficient to convert the SL-CoV S from non-ACE2 binding to human ACE2 binding, indicating that the SL-CoV S is largely compatible with SARS-CoV S protein both in structure and in function. The significance of these findings in relation to virus origin, virus recombination, and host switching is discussed….
    However, when the RBD of SL-CoV S was replaced with that from the SARS-CoV S, the hybrid S protein was able to use the huACE2 for cell entry, implying that the SL-CoV S proteins are structurally and functionally very similar to the SARS-CoV S. These results suggest that although the SL-CoVs discovered in bats so far are unlikely to infect humans using ACE2 as a receptor, it remains to be seen whether they are able to use other surface molecules of certain human cell types to gain entry. It is also conceivable that these viruses may become infectious to humans if they undergo N-terminal sequence variation, for example, through recombination with other CoVs, which in turn might lead to a productive interaction with ACE2 or other surface proteins on human cells. https://jvi.asm.org/content/82/4/1899
    ………………………………………..….
    J Virol. 2008 Feb; 82(4): 1899–1907.
    Published online 2007 Dec 12.
    Difference in Receptor Usage between Severe Acute Respiratory Syndrome (SARS) Coronavirus and SARS-Like Coronavirus of Bat Origin▿
    Wuze Ren,1,† Xiuxia Qu,2,† Wendong Li,1,‡ Zhenggang Han,1 Meng Yu,3 Peng Zhou,1 Shu-Yi Zhang,4 Lin-Fa Wang,3,* Hongkui Deng,2 and Zhengli Shi1,*

    Angiotensin-converting enzyme 2 (ACE2) has been identified as the functional receptor of SARS-CoV, and the molecular interaction between ACE2 and the SARS-CoV S protein has been well characterized (27, 28, 31, 42). A 193-residue fragment (amino acids [aa] 318 to 510) in the SARS-CoV S protein was demonstrated to be the minimal receptor-binding domain (RBD) which alone was able to efficiently bind to ACE2 (1, 42a, 45). Furthermore, it was shown that minor changes in amino acid residues of the receptor-binding motif (RBM) of SARS-CoV S protein could abolish the entry of SARS-CoV into cells expressing human ACE2 (huACE2) (7, 31). In the corresponding RBD region of the SL-CoV S proteins, there is significant sequence divergence from those of the SARS-CoV S proteins, including two deletions of 5 and 12 or 13 aa. From crystal-structural analysis of the S-ACE2 complex, it was predicted that the S protein of SL-CoV is unlikely to use huACE2 as an entry receptor (30), although this has never been experimentally proven due to the lack of live SL-CoV isolates. Whether it is possible to construct an ACE2-binding SL-CoV S protein by replacing the RBD with that from SARS-CoV S proteins is also unknown.
    https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2258702/

    51

  • #
    James Poulos

    If China had said nothing at all we’d all have put it down to another bad flu season… now all of a sudden every possible symptom of every possible disease is attributed to COVID 19.

    COVID Fear is beginning to feel like CLIMATE Fear.

    377

    • #
      Sceptical Sam

      Oh dear me!

      They’re out en masse today.

      They must have over done the Kool-aid last night.

      Your inability to discriminate between two different issues is a sign of your intellectual weakness.

      Fear has nothing to do with it. It’s an emotion.

      37

    • #
      rowingboat

      I live in a part of Sydney which is ground zero of Chinese immigration, near Eastwood. In mid-to-late February during my evening jogs around the oval, my fellow Chinese exercisers scattered like pigeons/seagulls would do as I approached them from behind (strange at the time, but clearly now they were social distancing from me).

      On the 3rd of March near lunchtime, Eastwood was deserted including restaurants which would normally be packed… the few women about were all wearing face masks but not all of the Chinese men interestingly enough; the lady who cut my hair that day was completely covered head to toe… face mask, gloves with only her eyes showing. That was the day I became scared and told my family they’re not going to the shops anymore.

      With all of the high rise apartments about us with often 3 generations of family living in the same home, if Covid-19 had taken hold where I live, we’ve have had an Italy/Spain situation on our hands. I continued with my weekly shop at Eastwood and in mid May, the stores and restaurants were returning back to normal.

      20

      • #
        Bill In Oz

        They were all plugged into the social media ‘news’ from China.
        They knew in late January long before it had entered many Australian’s consciousness.

        43

  • #
    Another Ian

    For the covid file

    “From the same site, but it was just a copy of this site:
    https://conservativewoman.co.uk/recovery-the-plot-sickens/

    The forces of evil in the HCQ story, incompetence, corruption, both?

    Professor Raoult in Marseille called it ‘monstrous’. Forty-one per cent of ventilated NHS patients died within 28 days; it’s 16 per cent in the Marseille ICU. Twenty-five per cent of NHS patients on oxygen support died; in Marseille only 5 per cent of hospitalised patients were lost. And 13 per cent of NHS patients not requiring breathing support still died, but only 0.6 per cent of patients treated in Marseille with Raoult’s hydroxychloroquine combination therapy.

    https://chiefio.wordpress.com/2020/05/28/w-o-o-d-28-may-2020/#comment-130777

    100

    • #
      Peter C

      Yes interesting. The British medical establishment is looking quite useless, if not positively evil.

      Note that the French reporter asks Prof Landry an insightful and probing question. If only more reporters did that.

      80

  • #
    Peter Fitzroy

    According to PerthNow, Coronavirus was circulating in Italy before the first cases were reported in China

    219

  • #
    Matty

    I’m told I will very likely have some lung scarring from childhood asthma and you could say it all leaves a mark. Chest infections, seasonal bugs, the stuff we inhale every day, age. Every other part of me is showing signs too and a chunk of these people will be smokers or with history of exposure to harmful things via their occupations – which until lately was half the male workforce.
    To see DR’s behaving like climate scientists is troubling indeed.

    121

    • #
      PeterW

      Me too. Never smoked, one asthma episode tgat sensitised me for some years, and chronically dusty environments over a working lifetime. There are signs.

      As for the initial topic. We have a virus with a known affinity for those with respiratory damage. Exactly what do they expect to find? Chicken…Egg… Correlation does not prove Causality.

      81

  • #
    Bill In Oz

    Jo, It’s good that you are still willing and able to look at the evidence
    On Covid 19 disease and report it here,
    Despite all the constant belittling from some persons
    Who want to persuade all here that the Covid 19 infectious pandemic
    With 300,000 people dead, is just a conspiracy.
    Talk about ignoring the evidence !

    311

    • #
      PeterS

      No one as far as I know is disputing the evidence of a pandemic. What’s in dispute is how to deal with it, and whether it’s enough to minimise its impact so we can return to some sort of normality in life. At the moment there are real fears of a second wave in spite of the restrictions. Time will tell if the second wave ends up as bad if not worse than the first. I hope not but we’ll know soon enough.

      132

      • #
        Bill In Oz

        Peter
        Some folk here have denied that this virus kills anybody
        ( It’s just co-morbidities and old age !!! )
        And some people here have denied that the Australian quarantines lock downs work.
        But the obvious facts are that when done properly they clearly work
        As demonstrated by all the Australian states except Victoria
        Where incompetence and . desire to conceal that incompetence,
        Have now resulted in the reimposition of a strong lock down.
        ( Eat your hearts out leftie BLM liberals !)
        However in South Australia, Tasmania, WA, Qld and even NSW
        This infectious disease is now under control and being eliminated.
        And life is already getting back to normal.
        Maybe Danny’s Viccies can get their act together as well, the second time around ?
        Maybe telling the public who are the infected ‘families’
        Who brought on the second lock down would help ?

        111

      • #
        Bill In Oz

        Peter each time I present the facts
        I get red thumbs for doing so.
        Lots of them – almost as many as Peter Fitzroy,
        our resident Global Warmist.
        The deniers are out in force on Jo’s blog
        I wonder why often.
        And there are two reasons that come to mind
        1: We have CCP sponsored trolls trying to disrupt the blog
        ( As made public by ScoMo on Friday )
        2: We have another band of trolls disrupting the blog
        To promote the Trump/Bolanaro agenda
        That this new infectious pandemic disease is ‘just a flu’.

        Actually probably both are ‘here’
        Some via commenting
        And some just to red thumb.

        35

    • #
      Matty

      Bill I don’t think anyone(that I’m aware of) said it was just a conspiracy, it’s an identifiable global bug but we have activist DR’s running the show who either don’t know or care that the economy is actually one big life support system. Especially in the global sense, we know that any downturn will kill millions of vulnerable so the first world went and engineered a downturn out of selfish fear. The UN of all people understand this – enough said about them. So much for “defeating the virus”. It was waiting for us.

      81

      • #
        Bill In Oz

        The answer was shutting down the airlines globally back in January.
        It’s the airlines of the world which spread this infectious pandemic disease.
        But that has never happened.
        TOO MANY VESTED INTERESTS TO PROTECT
        Those countries which shut down the airlines have done far better.
        EG New Zealand, Australia, Taiwan, the Pacific Island nations
        ( Even China !! )
        Those nations which ignored this basic protective measure have suffered massively
        And that includes places like Brazil, Nigeria etc.

        57

    • #
      Rob Kennedy

      Yes, the pandemic has killed 466728 (according to worldometer a few minutes ago). The Kong Kong flu 1968 – 1970 killed 1 million + ( I was off work for 3 weeks and sick as a dog for 2 weeks). The history of pandemics is easy to check online. What a lot of people see, however, is that previously governments did not shut down their economies to try to mitigate the effects. The scenario in 2020 is reminiscent of the Vietnam War tagline when viewing the smoking ruins of someone’s village, “We had to destroy the village to save it.”, or the spraying of ‘Agent Orange’ on the jungle so that they could see the bad guys.
      The destruction of businesses will be far more catastrophic than the pandemic.
      Many people are more optimistic it must be said, especially those allied to the aims of the World Econonic Forum. They are planning for “The Great Reset” of capitalism.
      “The changes we have already seen in response to COVID-19 prove that a reset of our economic and social foundations is possible.”
      “To achieve a better outcome, the world must act jointly and swiftly to revamp all aspects of our societies and economies, from education to social contracts and working conditions. Every country, from the United States to China, must participate, and every industry, from oil and gas to tech, must be transformed. In short, we need a “Great Reset” of capitalism.”
      In one of their videos Klaus Schwab says. “We have to live up to the expectations which we have created.” (emphasis mine)
      When you peruse their website (https://www.weforum.org/) you realise how deeply linked are the desired outcomes of the Climate Change Hoax and the pandemic.
      I do not agree with the globalists plans, but that’s probably because I’m just a “deplorable”.

      41

      • #
        Rolf

        It’s funny. The flu kills so many but it’s damned hard to find any doctor saying he/she had a patient dying from the flu. I can’t find one can you ? With 10.000 deads every year it should be so easy to find in every hospital ? But no.

        01

      • #
        TedM

        Yes Rob it is claimed that the Hong Kong flu killed over 1 million people. However there was not the same response to that flu, no social distancing, no lock downs and not the same travel restrictions, not to add that this pandemic has only been running outside of China for a few months.

        So please do not make inappropriate comparisons.

        00

  • #
    Plain Jane

    [Duplicate]AD

    00

  • #
    Plain Jane

    MR MODERATOR, CAN YOU PLEASE DELETE THE FIRST POST AS IT WAS INCOMPLETE AND PUT IN THIS ONE

    On the topic of the virus circulating in Italy very early that Peter Fitzroy brought up, there is a blog post by an anonymous, that I read in February sometime, but was posted on 31st January. Either this bloke had a crystal ball, or he had the connections he said he did. He had knowledge of the coming outbreak in Italy at a time when from the official numbers, he could not have surmised that. Remember, this post is from 31st January 2020. ….”The only reason you’re not already seeing thousands of people confirmed outside of China yet is because the incubation period but believe me the WHO is already talking about how “problematic” modeling the Chinese response in Western countries is going to be, and the first country they want to try it out in is Italy. If it begins a large outbreak in a major Italian city they want to work through the Italian authorities and world health organizations to begin locking down Italian cities in a vain attempt to slow down the spread at least until they can develop and distribute vaccines, which btw is where you need to start investing.”….. This is from an investment forum. The comment “and the first country they want to try it out in is Italy….. which suggests we are just guinea pigs.

    https://archive.4plebs.org/pol/thread/241674007

    When I read in February that the drug companies had already produced millions and millions of doses of chloroquine (already produced the stuff before the pandemic was official) I thought it highly likely that Chloroquine was effective, at least to some extent. Drug companies are not stupid and are major players and, along with armament producers, are bigger players than most governments.

    I am horrified by the response of many governments, including ours (OZ), in blocking a medication, chloroquine, that is likely to help so many millions of people from suffering. And even if it is not effective, it is not very harmful and is known not to be for 70 yrs or more. I get the political “Orange Man Bad” therefore many people have to die unnecessarily in the USA. I dont get why the UK wants to kill off its population so much, they could hardly have handled it worse, locking people in their homes but leaving borders open. Other than over the last few decades the UK has become a horrible totalitarian state. I do NOT know why the Australian government is blocking the use of CQ. My nurse friend from central Vic said they usually keep 6 bottles of CQ on the shelf in the hospital pharmacy and it all disappeared by early March, not to return, and other than give O2 or put on a ventilator there is no drug treatment for Cov patients planned in her hospital. Now I believe we have restricted prescribing of it to certain specialists only in OZ or NSW, could be wrong there. We don’t have “Orange Man Bad” in Oz to defeat so why??? Unless it is pressure from Big Pharma.

    It is almost like it was a designer virus, let go complicitly by governments all over the world, used for the various different political advantages in each country, for the benefit of Big Pharma, the Banks, Big Government in general, and with an evil and callous disregard for human life, and with the outcome of reducing human freedom, if not the population, and giving Big Government something ELSE to blame for the financial crash, other than their own wanton money printing (also known as stealing from the masses) of the last some decades. I knew they were never going to honour the promise to pay compulsory collected superannuation, just was never sure how “They” were going to do that.

    71

  • #
    Peter Fitzroy

    Reports show that nearly ½ of the inbound (to Australia) coronavirus cases come from 2 countries.

    A Pakistan and Great Britain
    B US and China

    Red for B, Green for A

    /tighten restrictions

    22

  • #
    Bill In Oz

    Sorry Jo but this needs to be stated and documented.

    Trump has worked out how to stop the pandemic in the USA.
    How ?
    Simple !
    Stop all testing for the Corona virus !
    “He gunner save hundreds of thousand os lives in the USA by doing this”
    https://www.abc.net.au/news/2020-06-21/trump-tulsa-reelection-rally-covid19-crowd-size/12377556
    Good luck with that ‘strategy’ my friends in the USA.

    17

  • #
    Dipole

    I wonder if lung damage in these asymptomatics shows up as lower blood oxygen readings ?

    30

  • #
    Kevin a

    CT scan before and after Covid would be more scientific
    Chronic respiratory diseases are very prevalent in Australia-an estimated 5.8 million Australians had at least one long-term respiratory condition in 2001.
    With about 5,400 deaths attributed to it in 2003, COPD is a major cause of death in Australia.

    1 in 20 Australians had COPD in 2017-18

    Japan is open. Taiwan is open, Vietnam is open.
    Victoria the banana republic, no vaccine will work against 20 strains of covid.

    https://www.thoracic.org.au/journal-publishing/command/download_file/id/21/filename/Respiratory_Infectious_Disease_Burden_in_Australia,_Edition_1,_March_2007_PDF.pdf

    https://www.aihw.gov.au/reports/chronic-respiratory-conditions/chronic-respiratory-diseases-australia/contents/summary

    51

  • #
    Kevin a

    Coronavirus is weakening, could die out on its own without a vaccine and patients now survive infections that would have killed them at start of the pandemic, claims Italian expert
    https://www.dailymail.co.uk/news/article-8444151/Coronavirus-withered-aggressive-tiger-wild-cat-Italian-scientist-claims.html

    11

  • #
    Aaron Christiansen

    No mention of “before x-rays” vs “after x-rays”?

    For the study, they examined data from 178 patients with PCR-confirmed SARS-CoV-2 infection in the Wanzhou District in China
    Lab tests show there was lung damage in just over half the group, inflammation (CRP), and there was some liver damage (ALT):

    149 men in the study.

    According to this report (2010): Male 15+ = 50% are smokers (page 2)

    Smoking damages the lungs and the liver.

    A quick search of the article for the word, “smoke” turns up no results, so I am guessing they did not control for that variable…

    Is this a classic case of “post hoc ergo propter hoc”?

    31

  • #
    AZ1971

    “Lung damage” in this case is temporary. The same thing (ground glass opacities, or GGO) happens with the flu. every. single. time. Every respiratory infection with an inflammatory response generates that.

    For those asymptomatics who suffer chronic respiratory damage, their condition is based on everything from smoking habits to age to genetic predisposition — very little of which one can control to mitigate their risk.

    10

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